Advances in Parasitology, Vol. 29 by J.R. Baker (ed.), R. Muller (ed.)

By J.R. Baker (ed.), R. Muller (ed.)

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Perhaps this further distinguishes them from the busoga organisms that cause acute disease in the Lake Victoria region. Three strain groups, with high PDs, also existed in East Africa and were principally linked with animal infections; they may, therefore, be versions of T. b. brucei. Kiboko was the nearest to the hypothetical ancestral organism, both in PD and by virtue of its primary location in central and southern Kenya. It was probably a specialization of the zambezi strain group, since it broke off from this late during cluster formation.

All contained populations originally isolated from 26 D . G. GODFREY, R. D . BAKER, L. R. RICKMAN A N D D . MEHLITZ patients in and around the Luangwa Valley in Zambia. A strong link with human infectivity existed, since the zymodemes contained 97% (185/191) of the populations originating from patients in Zambia; similar populations were isolated from animals in the same collecting area. , 33 in 2 2 , 2 2 in 2204, and I7 in 23). Twenty of the unrecognized zymodemes from Zambia were in the sindo (Section IV B 2) or kakumbi (Section IV B 4) strain groups.

1983) was, in error, not included in our analyses. However, it differed from 258 and 264 in PEPl only, and so would probably have been placed alongside these in set 18. Enzyme profiles of isolates from camels in Sudan displayed similar combinations of the variant enzymes used in common with this study (Boid, 1988), while further enzymes revealed additional polymorphisms. Although a precise comparison between the two studies is not easy, because the patterns for ALAT and PEPl do not entirely correspond, Boid’s finding of several forms of ALAT and ME indicates further genetic variation in T.

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